[{"type":"journal_article","publication":"Psychophysiology","status":"public","abstract":[{"text":"<jats:title>Abstract</jats:title><jats:p>For motor learning, the processing of behavioral outcomes is of high significance. The feedback‐related negativity (FRN) is an event‐related potential, which is often described as a correlate of the reward prediction error in reinforcement learning. The number of studies examining the FRN in motor tasks is increasing. This meta‐analysis summarizes the component in the motor domain and compares it to the cognitive domain. Therefore, a data set of a previous meta‐analysis in the cognitive domain that comprised 47 studies  was reanalyzed and compared to additional 25 studies of the motor domain. Further, a moderator analysis for the studies in the motor domain was conducted. The FRN amplitude was higher in the motor domain than in the cognitive domain. This might be related to a higher task complexity and a higher feedback ambiguity of motor tasks. The FRN latency was shorter in the motor domain than in the cognitive domain. Given that sensory information can be used as an external feedback predictor prior to the presentation of the final feedback, reward processing in the motor domain may have been faster and reduced the FRN latency. The moderator variable analysis revealed that the feedback modality influenced the FRN latency, with shorter FRN latencies after bimodal than after visual feedback. Processing of outcome feedback seems to share basic principles in both domains; however, differences exist and should be considered in FRN studies. Future research is motivated to scrutinize the effects of bimodal feedback and other moderators within the motor domain.</jats:p>","lang":"eng"}],"user_id":"668","_id":"48715","language":[{"iso":"eng"}],"keyword":["Experimental and Cognitive Psychology","Neuropsychology and Physiological Psychology","Biological Psychiatry","Cognitive Neuroscience","Developmental Neuroscience","Endocrine and Autonomic Systems","Neurology","Experimental and Cognitive Psychology","Neuropsychology and Physiological Psychology","General Neuroscience"],"issue":"12","publication_status":"published","publication_identifier":{"issn":["0048-5772","1469-8986"]},"citation":{"mla":"Faßbender, Laura, et al. “Feedback Processing in Cognitive and Motor Tasks: A Meta‐analysis on the Feedback‐related Negativity.” <i>Psychophysiology</i>, vol. 60, no. 12, Wiley, 2023, doi:<a href=\"https://doi.org/10.1111/psyp.14439\">10.1111/psyp.14439</a>.","bibtex":"@article{Faßbender_Krause_Weigelt_2023, title={Feedback processing in cognitive and motor tasks: A meta‐analysis on the feedback‐related negativity}, volume={60}, DOI={<a href=\"https://doi.org/10.1111/psyp.14439\">10.1111/psyp.14439</a>}, number={12}, journal={Psychophysiology}, publisher={Wiley}, author={Faßbender, Laura and Krause, Daniel and Weigelt, Matthias}, year={2023} }","short":"L. Faßbender, D. Krause, M. Weigelt, Psychophysiology 60 (2023).","apa":"Faßbender, L., Krause, D., &#38; Weigelt, M. (2023). Feedback processing in cognitive and motor tasks: A meta‐analysis on the feedback‐related negativity. <i>Psychophysiology</i>, <i>60</i>(12). <a href=\"https://doi.org/10.1111/psyp.14439\">https://doi.org/10.1111/psyp.14439</a>","chicago":"Faßbender, Laura, Daniel Krause, and Matthias Weigelt. “Feedback Processing in Cognitive and Motor Tasks: A Meta‐analysis on the Feedback‐related Negativity.” <i>Psychophysiology</i> 60, no. 12 (2023). <a href=\"https://doi.org/10.1111/psyp.14439\">https://doi.org/10.1111/psyp.14439</a>.","ieee":"L. Faßbender, D. Krause, and M. Weigelt, “Feedback processing in cognitive and motor tasks: A meta‐analysis on the feedback‐related negativity,” <i>Psychophysiology</i>, vol. 60, no. 12, 2023, doi: <a href=\"https://doi.org/10.1111/psyp.14439\">10.1111/psyp.14439</a>.","ama":"Faßbender L, Krause D, Weigelt M. Feedback processing in cognitive and motor tasks: A meta‐analysis on the feedback‐related negativity. <i>Psychophysiology</i>. 2023;60(12). doi:<a href=\"https://doi.org/10.1111/psyp.14439\">10.1111/psyp.14439</a>"},"intvolume":"        60","year":"2023","date_created":"2023-11-08T20:37:11Z","author":[{"last_name":"Faßbender","full_name":"Faßbender, Laura","first_name":"Laura"},{"first_name":"Daniel","last_name":"Krause","orcid":"orcid.org/0000-0001-5391-885X","full_name":"Krause, Daniel","id":"668"},{"first_name":"Matthias","full_name":"Weigelt, Matthias","id":"36388","last_name":"Weigelt"}],"volume":60,"date_updated":"2023-11-08T21:10:01Z","publisher":"Wiley","doi":"10.1111/psyp.14439","title":"Feedback processing in cognitive and motor tasks: A meta‐analysis on the feedback‐related negativity"},{"publication_status":"published","publication_identifier":{"issn":["2158-3188"]},"issue":"1","year":"2022","citation":{"apa":"Zheng, Y., Rajcsanyi, L. S., Herpertz-Dahlmann, B., Seitz, J., de Zwaan, M., Herzog, W., Ehrlich, S., Zipfel, S., Giel, K., Egberts, K., Burghardt, R., Föcker, M., Al-Lahham, S., Peters, T., Libuda, L., Antel, J., Hebebrand, J., &#38; Hinney, A. (2022). PTBP2 – a gene with relevance for both Anorexia nervosa and body weight regulation. <i>Translational Psychiatry</i>, <i>12</i>(1), Article 241. <a href=\"https://doi.org/10.1038/s41398-022-02018-5\">https://doi.org/10.1038/s41398-022-02018-5</a>","short":"Y. Zheng, L.S. Rajcsanyi, B. Herpertz-Dahlmann, J. Seitz, M. de Zwaan, W. Herzog, S. Ehrlich, S. Zipfel, K. Giel, K. Egberts, R. Burghardt, M. Föcker, S. Al-Lahham, T. Peters, L. Libuda, J. Antel, J. Hebebrand, A. Hinney, Translational Psychiatry 12 (2022).","mla":"Zheng, Yiran, et al. “PTBP2 – a Gene with Relevance for Both Anorexia Nervosa and Body Weight Regulation.” <i>Translational Psychiatry</i>, vol. 12, no. 1, 241, Springer Science and Business Media LLC, 2022, doi:<a href=\"https://doi.org/10.1038/s41398-022-02018-5\">10.1038/s41398-022-02018-5</a>.","bibtex":"@article{Zheng_Rajcsanyi_Herpertz-Dahlmann_Seitz_de Zwaan_Herzog_Ehrlich_Zipfel_Giel_Egberts_et al._2022, title={PTBP2 – a gene with relevance for both Anorexia nervosa and body weight regulation}, volume={12}, DOI={<a href=\"https://doi.org/10.1038/s41398-022-02018-5\">10.1038/s41398-022-02018-5</a>}, number={1241}, journal={Translational Psychiatry}, publisher={Springer Science and Business Media LLC}, author={Zheng, Yiran and Rajcsanyi, Luisa Sophie and Herpertz-Dahlmann, Beate and Seitz, Jochen and de Zwaan, Martina and Herzog, Wolfgang and Ehrlich, Stefan and Zipfel, Stephan and Giel, Katrin and Egberts, Karin and et al.}, year={2022} }","ieee":"Y. Zheng <i>et al.</i>, “PTBP2 – a gene with relevance for both Anorexia nervosa and body weight regulation,” <i>Translational Psychiatry</i>, vol. 12, no. 1, Art. no. 241, 2022, doi: <a href=\"https://doi.org/10.1038/s41398-022-02018-5\">10.1038/s41398-022-02018-5</a>.","chicago":"Zheng, Yiran, Luisa Sophie Rajcsanyi, Beate Herpertz-Dahlmann, Jochen Seitz, Martina de Zwaan, Wolfgang Herzog, Stefan Ehrlich, et al. “PTBP2 – a Gene with Relevance for Both Anorexia Nervosa and Body Weight Regulation.” <i>Translational Psychiatry</i> 12, no. 1 (2022). <a href=\"https://doi.org/10.1038/s41398-022-02018-5\">https://doi.org/10.1038/s41398-022-02018-5</a>.","ama":"Zheng Y, Rajcsanyi LS, Herpertz-Dahlmann B, et al. PTBP2 – a gene with relevance for both Anorexia nervosa and body weight regulation. <i>Translational Psychiatry</i>. 2022;12(1). doi:<a href=\"https://doi.org/10.1038/s41398-022-02018-5\">10.1038/s41398-022-02018-5</a>"},"intvolume":"        12","publisher":"Springer Science and Business Media LLC","date_updated":"2022-10-26T15:49:24Z","date_created":"2022-07-06T15:14:13Z","author":[{"full_name":"Zheng, Yiran","last_name":"Zheng","first_name":"Yiran"},{"full_name":"Rajcsanyi, Luisa Sophie","last_name":"Rajcsanyi","first_name":"Luisa Sophie"},{"last_name":"Herpertz-Dahlmann","full_name":"Herpertz-Dahlmann, Beate","first_name":"Beate"},{"full_name":"Seitz, Jochen","last_name":"Seitz","first_name":"Jochen"},{"first_name":"Martina","full_name":"de Zwaan, Martina","last_name":"de Zwaan"},{"first_name":"Wolfgang","full_name":"Herzog, Wolfgang","last_name":"Herzog"},{"last_name":"Ehrlich","full_name":"Ehrlich, Stefan","first_name":"Stefan"},{"full_name":"Zipfel, Stephan","last_name":"Zipfel","first_name":"Stephan"},{"last_name":"Giel","full_name":"Giel, Katrin","first_name":"Katrin"},{"first_name":"Karin","full_name":"Egberts, Karin","last_name":"Egberts"},{"last_name":"Burghardt","full_name":"Burghardt, Roland","first_name":"Roland"},{"last_name":"Föcker","full_name":"Föcker, Manuel","first_name":"Manuel"},{"first_name":"Saad","full_name":"Al-Lahham, Saad","last_name":"Al-Lahham"},{"first_name":"Triinu","last_name":"Peters","full_name":"Peters, Triinu"},{"orcid":"0000-0003-1603-3133","last_name":"Libuda","id":"88682","full_name":"Libuda, Lars","first_name":"Lars"},{"full_name":"Antel, Jochen","last_name":"Antel","first_name":"Jochen"},{"last_name":"Hebebrand","full_name":"Hebebrand, Johannes","first_name":"Johannes"},{"first_name":"Anke","last_name":"Hinney","full_name":"Hinney, Anke"}],"volume":12,"title":"PTBP2 – a gene with relevance for both Anorexia nervosa and body weight regulation","doi":"10.1038/s41398-022-02018-5","type":"journal_article","publication":"Translational Psychiatry","abstract":[{"text":"<jats:title>Abstract</jats:title><jats:p>Genetic factors are relevant for both eating disorders and body weight regulation. A recent genome-wide association study (GWAS) for anorexia nervosa (AN) detected eight genome-wide significant chromosomal loci. One of these loci, rs10747478, was also genome-wide and significantly associated with body mass index (BMI). The nearest coding gene is the Polypyrimidine Tract Binding Protein 2 gene (<jats:italic>PTBP2</jats:italic>). To detect mutations in <jats:italic>PTBP2</jats:italic>, Sanger sequencing of the coding region was performed in 192 female patients with AN (acute or recovered) and 191 children or adolescents with (extreme) obesity. Twenty-five variants were identified. Twenty-three of these were predicted to be pathogenic or functionally relevant in at least one in silico tool. Two novel synonymous variants (p.Ala77Ala and p.Asp195Asp), one intronic SNP (rs188987764), and the intronic deletion (rs561340981) located in the highly conserved region of <jats:italic>PTBP2</jats:italic> may have functional consequences. Ten of 20 genes interacting with <jats:italic>PTBP2</jats:italic> were studied for their impact on body weight regulation based on either previous functional studies or GWAS hits for body weight or BMI. In a GWAS for BMI (Pulit et al. 2018), the number of genome-wide significant associations at the <jats:italic>PTBP2</jats:italic> locus was different between males (60 variants) and females (two variants, one of these also significant in males). More than 65% of these 61 variants showed differences in the effect size pertaining to BMI between sexes (absolute value of <jats:italic>Z</jats:italic>-score &gt;2, two-sided <jats:italic>p</jats:italic> &lt; 0.05). One LD block overlapping 5′UTR and all coding regions of <jats:italic>PTBP2</jats:italic> comprises 56 significant variants in males. The analysis based on sex-stratified BMI GWAS summary statistics implies that <jats:italic>PTBP2</jats:italic> may have a more pronounced effect on body weight regulation in males than in females.</jats:p>","lang":"eng"}],"status":"public","_id":"32326","user_id":"88682","department":[{"_id":"35"}],"article_number":"241","keyword":["Biological Psychiatry","Cellular and Molecular Neuroscience","Psychiatry and Mental health"],"language":[{"iso":"eng"}]},{"publisher":"Springer Science and Business Media LLC","date_created":"2022-07-06T15:14:39Z","title":"The adrenal steroid profile in adolescent depression: a valuable bio-readout?","issue":"1","year":"2022","keyword":["Biological Psychiatry","Cellular and Molecular Neuroscience","Psychiatry and Mental health"],"language":[{"iso":"eng"}],"publication":"Translational Psychiatry","abstract":[{"text":"<jats:title>Abstract</jats:title><jats:p>There is preliminary evidence that adrenal steroids other than cortisol may be valuable biomarkers for major depressive disorder (MDD). So far, studies have been conducted in adults only, and conclusions are limited, mainly due to small sample sizes. Therefore, the present study assessed whether adrenal steroids serve as biomarkers for adolescent MDD. In 261 depressed adolescents (170 females) treated at a single psychiatric hospital, serum adrenal steroids (progesterone, 17-hydroxyprogesterone, 21-deoxycortisol, 11-deoxycortisol, cortisol, cortisone, deoxycorticosterone, corticosterone) were determined by liquid chromatography-tandem mass spectrometry. Findings were compared to that of an age- and sex-matched reference cohort (<jats:italic>N</jats:italic> = 255) by nonparametric analysis of variance. Nonparametric receiver operating characteristics (ROC) analyses were conducted to evaluate the diagnostic performance of single steroids and steroid ratios to classify depression status. Sensitivity analyses considered important confounders of adrenal functioning, and ROC results were verified by cross-validation. Compared to the reference cohort, levels of deoxycorticosterone and 21-deoxycortisol were decreased (<jats:italic>P</jats:italic> &lt; 0.001). All other glucocorticoid- and mineralocorticoid-related steroids were increased (<jats:italic>P</jats:italic> &lt; 0.001). The corticosterone to deoxycorticosterone ratio evidenced excellent classification characteristics, especially in females (AUC: 0.957; sensitivity: 0.902; specificity: 0.891). The adrenal steroid metabolome qualifies as a bio-readout reflecting adolescent MDD by a distinct steroid pattern that indicates dysfunction of the hypothalamus–pituitary–adrenal axis. Moreover, the corticosterone to deoxycorticosterone ratio may prospectively qualify to contribute to precision medicine in psychiatry by identifying those patients who might benefit from antiglucocorticoid treatment or those at risk for recurrence when adrenal dysfunction has not resolved.</jats:p>","lang":"eng"}],"date_updated":"2023-01-06T11:48:51Z","volume":12,"author":[{"full_name":"Hirtz, Raphael","last_name":"Hirtz","first_name":"Raphael"},{"last_name":"Libuda","orcid":"0000-0003-1603-3133","id":"88682","full_name":"Libuda, Lars","first_name":"Lars"},{"last_name":"Hinney","full_name":"Hinney, Anke","first_name":"Anke"},{"full_name":"Föcker, Manuel","last_name":"Föcker","first_name":"Manuel"},{"first_name":"Judith","last_name":"Bühlmeier","full_name":"Bühlmeier, Judith","id":"89838"},{"first_name":"Paul-Martin","last_name":"Holterhus","full_name":"Holterhus, Paul-Martin"},{"last_name":"Kulle","full_name":"Kulle, Alexandra","first_name":"Alexandra"},{"full_name":"Kiewert, Cordula","last_name":"Kiewert","first_name":"Cordula"},{"first_name":"Berthold P.","last_name":"Hauffa","full_name":"Hauffa, Berthold P."},{"first_name":"Johannes","full_name":"Hebebrand, Johannes","last_name":"Hebebrand"},{"last_name":"Grasemann","full_name":"Grasemann, Corinna","first_name":"Corinna"}],"doi":"10.1038/s41398-022-01966-2","publication_identifier":{"issn":["2158-3188"]},"publication_status":"published","intvolume":"        12","citation":{"chicago":"Hirtz, Raphael, Lars Libuda, Anke Hinney, Manuel Föcker, Judith Bühlmeier, Paul-Martin Holterhus, Alexandra Kulle, et al. “The Adrenal Steroid Profile in Adolescent Depression: A Valuable Bio-Readout?” <i>Translational Psychiatry</i> 12, no. 1 (2022). <a href=\"https://doi.org/10.1038/s41398-022-01966-2\">https://doi.org/10.1038/s41398-022-01966-2</a>.","ieee":"R. Hirtz <i>et al.</i>, “The adrenal steroid profile in adolescent depression: a valuable bio-readout?,” <i>Translational Psychiatry</i>, vol. 12, no. 1, Art. no. 255, 2022, doi: <a href=\"https://doi.org/10.1038/s41398-022-01966-2\">10.1038/s41398-022-01966-2</a>.","ama":"Hirtz R, Libuda L, Hinney A, et al. The adrenal steroid profile in adolescent depression: a valuable bio-readout? <i>Translational Psychiatry</i>. 2022;12(1). doi:<a href=\"https://doi.org/10.1038/s41398-022-01966-2\">10.1038/s41398-022-01966-2</a>","apa":"Hirtz, R., Libuda, L., Hinney, A., Föcker, M., Bühlmeier, J., Holterhus, P.-M., Kulle, A., Kiewert, C., Hauffa, B. P., Hebebrand, J., &#38; Grasemann, C. (2022). The adrenal steroid profile in adolescent depression: a valuable bio-readout? <i>Translational Psychiatry</i>, <i>12</i>(1), Article 255. <a href=\"https://doi.org/10.1038/s41398-022-01966-2\">https://doi.org/10.1038/s41398-022-01966-2</a>","short":"R. Hirtz, L. Libuda, A. Hinney, M. Föcker, J. Bühlmeier, P.-M. Holterhus, A. Kulle, C. Kiewert, B.P. Hauffa, J. Hebebrand, C. Grasemann, Translational Psychiatry 12 (2022).","mla":"Hirtz, Raphael, et al. “The Adrenal Steroid Profile in Adolescent Depression: A Valuable Bio-Readout?” <i>Translational Psychiatry</i>, vol. 12, no. 1, 255, Springer Science and Business Media LLC, 2022, doi:<a href=\"https://doi.org/10.1038/s41398-022-01966-2\">10.1038/s41398-022-01966-2</a>.","bibtex":"@article{Hirtz_Libuda_Hinney_Föcker_Bühlmeier_Holterhus_Kulle_Kiewert_Hauffa_Hebebrand_et al._2022, title={The adrenal steroid profile in adolescent depression: a valuable bio-readout?}, volume={12}, DOI={<a href=\"https://doi.org/10.1038/s41398-022-01966-2\">10.1038/s41398-022-01966-2</a>}, number={1255}, journal={Translational Psychiatry}, publisher={Springer Science and Business Media LLC}, author={Hirtz, Raphael and Libuda, Lars and Hinney, Anke and Föcker, Manuel and Bühlmeier, Judith and Holterhus, Paul-Martin and Kulle, Alexandra and Kiewert, Cordula and Hauffa, Berthold P. and Hebebrand, Johannes and et al.}, year={2022} }"},"_id":"32327","department":[{"_id":"35"}],"user_id":"88682","article_number":"255","type":"journal_article","status":"public"},{"abstract":[{"text":"<jats:p>Whereas initial findings have already identified cortical patterns accompanying proprioceptive deficiencies in patients after anterior cruciate ligament reconstruction (ACLR), little is known about compensatory sensorimotor mechanisms for re-establishing postural control. Therefore, the aim of the present study was to explore leg dependent patterns of cortical contributions to postural control in patients 6 weeks following ACLR. A total of 12 patients after ACLR (25.1 ± 3.2 years, 178.1 ± 9.7 cm, 77.5 ± 14.4 kg) and another 12 gender, age, and activity matched healthy controls participated in this study. All subjects performed 10 × 30 s. single leg stances on each leg, equipped with 64-channel mobile electroencephalography (EEG). Postural stability was quantified by area of sway and sway velocity. Estimations of the weighted phase lag index were conducted as a cortical measure of functional connectivity. The findings showed significant group × leg interactions for increased functional connectivity in the anterior cruciate ligament (ACL) injured leg, predominantly including fronto−parietal [<jats:italic>F</jats:italic><jats:sub>(1, 22)</jats:sub> = 8.41, <jats:italic>p</jats:italic> ≤ 0.008, η<jats:sup>2</jats:sup> = 0.28], fronto−occipital [<jats:italic>F</jats:italic><jats:sub>(1, 22)</jats:sub> = 4.43, <jats:italic>p</jats:italic> ≤ 0.047, η<jats:sup>2</jats:sup> = 0.17], parieto−motor [<jats:italic>F</jats:italic><jats:sub>(1, 22)</jats:sub> = 10.30, <jats:italic>p</jats:italic> ≤ 0.004, η<jats:sup>2</jats:sup> = 0.32], occipito−motor [<jats:italic>F</jats:italic><jats:sub>(1, 22)</jats:sub> = 5.21, <jats:italic>p</jats:italic> ≤ 0.032, η<jats:sup>2</jats:sup> = 0.19], and occipito−parietal [<jats:italic>F</jats:italic><jats:sub>(1, 22)</jats:sub> = 4.60, <jats:italic>p</jats:italic> ≤ 0.043, η<jats:sup>2</jats:sup> = 0.17] intra−hemispherical connections in the contralateral hemisphere and occipito−motor [<jats:italic>F</jats:italic><jats:sub>(1, 22)</jats:sub> = 7.33, <jats:italic>p</jats:italic> ≤ 0.013, η<jats:sup>2</jats:sup> = 0.25] on the ipsilateral hemisphere to the injured leg. Higher functional connectivity in patients after ACLR, attained by increased emphasis of functional connections incorporating the somatosensory and visual areas, may serve as a compensatory mechanism to control postural stability of the injured leg in the early phase of rehabilitation. These preliminary results may help to develop new neurophysiological assessments for detecting functional deficiencies after ACLR in the future.</jats:p>","lang":"eng"}],"status":"public","publication":"Frontiers in Human Neuroscience","type":"journal_article","keyword":["Behavioral Neuroscience","Biological Psychiatry","Psychiatry and Mental health","Neurology","Neuropsychology and Physiological Psychology"],"language":[{"iso":"eng"}],"_id":"32434","department":[{"_id":"17"},{"_id":"172"}],"user_id":"46","year":"2021","intvolume":"        15","citation":{"apa":"Lehmann, T., Büchel, D., Mouton, C., Gokeler, A., Seil, R., &#38; Baumeister, J. (2021). Functional Cortical Connectivity Related to Postural Control in Patients Six Weeks After Anterior Cruciate Ligament Reconstruction. <i>Frontiers in Human Neuroscience</i>, <i>15</i>. <a href=\"https://doi.org/10.3389/fnhum.2021.655116\">https://doi.org/10.3389/fnhum.2021.655116</a>","short":"T. Lehmann, D. Büchel, C. Mouton, A. Gokeler, R. Seil, J. Baumeister, Frontiers in Human Neuroscience 15 (2021).","mla":"Lehmann, Tim, et al. “Functional Cortical Connectivity Related to Postural Control in Patients Six Weeks After Anterior Cruciate Ligament Reconstruction.” <i>Frontiers in Human Neuroscience</i>, vol. 15, Frontiers Media SA, 2021, doi:<a href=\"https://doi.org/10.3389/fnhum.2021.655116\">10.3389/fnhum.2021.655116</a>.","bibtex":"@article{Lehmann_Büchel_Mouton_Gokeler_Seil_Baumeister_2021, title={Functional Cortical Connectivity Related to Postural Control in Patients Six Weeks After Anterior Cruciate Ligament Reconstruction}, volume={15}, DOI={<a href=\"https://doi.org/10.3389/fnhum.2021.655116\">10.3389/fnhum.2021.655116</a>}, journal={Frontiers in Human Neuroscience}, publisher={Frontiers Media SA}, author={Lehmann, Tim and Büchel, Daniel and Mouton, Caroline and Gokeler, Alli and Seil, Romain and Baumeister, Jochen}, year={2021} }","ieee":"T. Lehmann, D. Büchel, C. Mouton, A. Gokeler, R. Seil, and J. Baumeister, “Functional Cortical Connectivity Related to Postural Control in Patients Six Weeks After Anterior Cruciate Ligament Reconstruction,” <i>Frontiers in Human Neuroscience</i>, vol. 15, 2021, doi: <a href=\"https://doi.org/10.3389/fnhum.2021.655116\">10.3389/fnhum.2021.655116</a>.","chicago":"Lehmann, Tim, Daniel Büchel, Caroline Mouton, Alli Gokeler, Romain Seil, and Jochen Baumeister. “Functional Cortical Connectivity Related to Postural Control in Patients Six Weeks After Anterior Cruciate Ligament Reconstruction.” <i>Frontiers in Human Neuroscience</i> 15 (2021). <a href=\"https://doi.org/10.3389/fnhum.2021.655116\">https://doi.org/10.3389/fnhum.2021.655116</a>.","ama":"Lehmann T, Büchel D, Mouton C, Gokeler A, Seil R, Baumeister J. Functional Cortical Connectivity Related to Postural Control in Patients Six Weeks After Anterior Cruciate Ligament Reconstruction. <i>Frontiers in Human Neuroscience</i>. 2021;15. doi:<a href=\"https://doi.org/10.3389/fnhum.2021.655116\">10.3389/fnhum.2021.655116</a>"},"publication_identifier":{"issn":["1662-5161"]},"publication_status":"published","title":"Functional Cortical Connectivity Related to Postural Control in Patients Six Weeks After Anterior Cruciate Ligament Reconstruction","doi":"10.3389/fnhum.2021.655116","publisher":"Frontiers Media SA","date_updated":"2023-03-13T15:20:11Z","volume":15,"date_created":"2022-07-27T07:47:56Z","author":[{"id":"41584","full_name":"Lehmann, Tim","last_name":"Lehmann","first_name":"Tim"},{"first_name":"Daniel","id":"41088","full_name":"Büchel, Daniel","last_name":"Büchel"},{"first_name":"Caroline","full_name":"Mouton, Caroline","last_name":"Mouton"},{"first_name":"Alli","full_name":"Gokeler, Alli","last_name":"Gokeler"},{"first_name":"Romain","full_name":"Seil, Romain","last_name":"Seil"},{"first_name":"Jochen","full_name":"Baumeister, Jochen","id":"46","last_name":"Baumeister","orcid":"0000-0003-2683-5826"}]},{"doi":"10.1111/psyp.13508","title":"Valence‐dependent brain potentials of processing augmented feedback in learning a complex arm movement sequence","author":[{"last_name":"Krause","orcid":"orcid.org/0000-0001-5391-885X","full_name":"Krause, Daniel","id":"668","first_name":"Daniel"},{"first_name":"Timo","full_name":"Koers, Timo","last_name":"Koers"},{"first_name":"Lisa Katharina","last_name":"Maurer","full_name":"Maurer, Lisa Katharina"}],"date_created":"2023-11-08T20:29:39Z","volume":57,"date_updated":"2023-11-08T21:13:52Z","publisher":"Wiley","citation":{"bibtex":"@article{Krause_Koers_Maurer_2019, title={Valence‐dependent brain potentials of processing augmented feedback in learning a complex arm movement sequence}, volume={57}, DOI={<a href=\"https://doi.org/10.1111/psyp.13508\">10.1111/psyp.13508</a>}, number={3}, journal={Psychophysiology}, publisher={Wiley}, author={Krause, Daniel and Koers, Timo and Maurer, Lisa Katharina}, year={2019} }","mla":"Krause, Daniel, et al. “Valence‐dependent Brain Potentials of Processing Augmented Feedback in Learning a Complex Arm Movement Sequence.” <i>Psychophysiology</i>, vol. 57, no. 3, Wiley, 2019, doi:<a href=\"https://doi.org/10.1111/psyp.13508\">10.1111/psyp.13508</a>.","short":"D. Krause, T. Koers, L.K. Maurer, Psychophysiology 57 (2019).","apa":"Krause, D., Koers, T., &#38; Maurer, L. K. (2019). Valence‐dependent brain potentials of processing augmented feedback in learning a complex arm movement sequence. <i>Psychophysiology</i>, <i>57</i>(3). <a href=\"https://doi.org/10.1111/psyp.13508\">https://doi.org/10.1111/psyp.13508</a>","chicago":"Krause, Daniel, Timo Koers, and Lisa Katharina Maurer. “Valence‐dependent Brain Potentials of Processing Augmented Feedback in Learning a Complex Arm Movement Sequence.” <i>Psychophysiology</i> 57, no. 3 (2019). <a href=\"https://doi.org/10.1111/psyp.13508\">https://doi.org/10.1111/psyp.13508</a>.","ieee":"D. Krause, T. Koers, and L. K. Maurer, “Valence‐dependent brain potentials of processing augmented feedback in learning a complex arm movement sequence,” <i>Psychophysiology</i>, vol. 57, no. 3, 2019, doi: <a href=\"https://doi.org/10.1111/psyp.13508\">10.1111/psyp.13508</a>.","ama":"Krause D, Koers T, Maurer LK. Valence‐dependent brain potentials of processing augmented feedback in learning a complex arm movement sequence. <i>Psychophysiology</i>. 2019;57(3). doi:<a href=\"https://doi.org/10.1111/psyp.13508\">10.1111/psyp.13508</a>"},"intvolume":"        57","year":"2019","issue":"3","publication_status":"published","publication_identifier":{"issn":["0048-5772","1469-8986"]},"language":[{"iso":"eng"}],"keyword":["Experimental and Cognitive Psychology","Neuropsychology and Physiological Psychology","Biological Psychiatry","Cognitive Neuroscience","Developmental Neuroscience","Endocrine and Autonomic Systems","Neurology","Experimental and Cognitive Psychology","Neuropsychology and Physiological Psychology","General Neuroscience"],"user_id":"668","_id":"48702","status":"public","abstract":[{"lang":"eng","text":"<jats:title>Abstract</jats:title><jats:p>ERPs in the EEG were scrutinized in learning a complex arm movement sequence with the aim to examine valence effects on processing augmented feedback during practice. Twenty‐four healthy subjects practiced one session with 192 feedback trials according to an adaptive bandwidth feedback approach with a high informational level of feedback information (i.e., amplitude and direction of errors). The bandwidth for successful performance (increase of a score for a monetary competition) was manipulated to yield a success rate (positive feedback frequency) of approximately 50% adaptive to the current performance level. This allowed a variation of feedback valence unconfounded by success rate. In line with our hypotheses, the EEG data showed a valence‐dependent feedback‐related negativity (FRN) and a later fronto‐central component at the FCz electrode as well as a P300 component at the Pz electrode. Moreover, the P300 and amplitudes in the FRN time window reduced in the second half of practice but were still dependent on feedback valence. Behavioral adjustments were larger after feedback with negative valence and were predicted by the late fronto‐central component. The data support the assumption of feedback valence‐dependent modulation of attentional cognitive involvement in motor control and learning.</jats:p>"}],"type":"journal_article","publication":"Psychophysiology"}]